Anti-DcR1 Antibody [TRAIL-R3-02] (FITC) (A86663)

Mouse monoclonal [TRAIL-R3-02] antibody to DcR1 (FITC) for Flow Cytometry.

Alternative Formats and ConjugatesAlternative Formats

Freight Charges
Lead Time
4-6 Business Days
Telephone
+1 (314) 370-6046
Mon - Fri, 8am - 4pm AST
Email
orders@antibodies.com
Name
Anti-DcR1 Antibody [TRAIL-R3-02] (FITC)
Description
Mouse monoclonal [TRAIL-R3-02] antibody to DcR1 (FITC).
Specificity
This antibody reacts with TRAIL-R3, a 35 kDa GPI-anchored extracellular membrane protein expressed mainly on neutrophils.
Applications
Flow Cytometry
Dilutions
Flow Cytometry: 1-5 µg/ml.
Reactivity
Human
Immunogen
Recombinant fusion protein of human IgG heavy chain and extracellular domain of TRAIL-R3 (amino acids 1-280).
Host
Mouse
Clonality
Monoclonal
Clone ID
TRAIL-R3-02
Isotype
IgG1
Conjugate

FITC

Excitation: 490nm, Emission: 525nm

Purification
This antibody is conjugated with Fluorescein Isothiocyanate (FITC) under optimum conditions. The conjugate is purified by size-exclusion chromatography; removing unconjugated antibody and free fluorochrome.
Concentration
100 µg/ml
Predicted MW
35 kDa
Product Form
Liquid
Formulation
Supplied in Phosphate Buffered Saline, pH 7.4, with 15 mM Sodium Azide.
Storage
Shipped at 4°C. Store in the dark at 2-8°C. Do not freeze!
Synonyms
Antagonist decoy receptor for TRAIL/Apo 2L, CD263, Cytotoxic TRAIL receptor 3, DCR1 TNFR, Decoy Receptor 1, Decoy TRAIL receptor without death domain, LIT, Lymphocyte inhibitor of TRAIL, MGC149501, MGC149502, TNF related apoptosis inducing ligand receptor 3, TNF-re ated apoptosis inducing ligand receptor 3, TNFRSF10C, TRAIL R3, TRAIL receptor 3, TRAILR3, TRID, Tumor necrosis factor receptor superfamily member 10C, Tumor necrosis factor receptor superfamily, member 10c, decoy without an intracellular domain
Disclaimer
This product is for research use only. It is not intended for diagnostic or therapeutic use.
Publishing research using Anti-DcR1 Antibody [TRAIL-R3-02] (FITC) (A86663)? Please let us know so that we can list the citation on this page.
Top